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FEAT-Chattanooga Now Listed Under IGIVE.COM
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doing your on-line Holiday Shopping this season,
remember to purchase through iGive.com and FEAT-Chattanooga will receive
a percentage of the sales.
WHAT IS IGIVE.COM:
iGive.com is the first and largest network of members, worthy causes,
online merchants, and Web sites, brought together to enable shoppers to list
and support their favorite worthy causes through online shopping.
HOW IT WORKS:
Charitably-minded individuals --Become
members.
(It’s free! Always!) Select FEAT
Chattanooga as your worthy cause to support. –Shop the mall
at iGive.com.
--Raise money for FEAT Chattanooga.
WHEN FEAT WILL RECEIVE CHECKS: iGive.com sends monthly checks to all verified
causes after they receive payment from affiliated merchants, 120 days in arrears.
The minimum for check issuance is $10.
If our cause has raised less than $10, our accrued funds will be rolled
over to the next pay period.
CauseHelp@iGive.com
http://www.igive.com/html/intro.cfm iGive.com, inc., 1740 Ridge Ave., Evanston,
IL 60201
The fine print from iGive.com
This e-mail
is sent to worthy causes listed at our site by iGive.com members. Every time our members shop at stores
in the Mall at iGive.com, a percentage of their purchases can
be donated to their favorite worthy causes. We offer a wide range of quality products
from top merchants – from CDs to books, office supplies, flowers, toys, and videos.
The causes listed at iGive.com are controlled by our members. They add their favorite causes to
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Researchers Disover Autism Gene
WASHINGTON
(AP) - Scientists have long theorized that about 16 different genes play in
who is born with the severe brain disorder autism - and now they've finally
found one of those genes.
A study of 57 autism patients found that 40 percent carry a mutated
version of the HOXA1 gene, which plays a
crucial role in early brain development. University
of Rochester scientists reported Monday.
Children need to inherit just one copy of the mutated gene from one parent
to have autism. In fact, scientists found only one patient, a very severe
case, who inherited a copy of the bad gene from both parents, suggesting that
when that first happens the fetus usually dies, said lead researcher Patircia
Rodier, who heads the university's National Institutes of Health - funded
autism research center.
The NIH called the finding a signigicant step in understanding what predisposes
people to developing autism. More than 400,000 Americans have the brain disorder,
characterized by profound social withdrawal, repetitive behavior and inability
to communicate. Research suggests it's caused when something goes wrong during
critical fetal brain development - a theory the gene discovery, in the December
issue of the journal Teratology, supports.
HOXA1 is one of a family of genese vital to early embryo development because
genes in the group turn on or off other genes. HOXA1's specific role is in
brain development. Mice who lack this gene have brainstem damage, malformed
ears and other classic signs of autism - one reason Rodier's research team
decided to check the gene's role in people.
It's not the kind of gene that could ever be fixed with gene therapy. But
the discovery may help doctors unravel just how the brain changes when HOXA1
is abnormal, Rodier said.
"If you figure out the brain changes, you're on your way, we hope, to finding
better treatments," she said.
On the Net:
NIH autism site: http://www.nichd.nih.gov/autism
Teratology Society site with link to study abstract: http://www.teratology.org/
SEE Page 4 FOR ANOTHER
AUTISM GENE ARTICLE
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