impressed current protection of a galvanized water tank

the white powder on car battery terminals is lead sulphate

shaping spark plug electrodes and indexing a briggs and stratton side valve engine for improved starting, fuel economy and power

rhyme as a non verbal therapy

dinosaurs and weight loss

the angiotensin converting enzyme and weight loss

waist to height ratio predictive of types 2 diabetes for women

body weight influenced by thousands of genes

the compendium and the BCD for weight loss





unbelievable !

lived 7





. .



nothing new about anorexia if you read that link,  all those lead poisoned from its ubiquitous historical use in  plumbing,  roofing,  false teeth,  medications,  the list is endless !


anorexia is a starving brain bringing the body into line.


from an enzymesandautism post  (10th july 04)  titled  xango executive board

MLM is a literal pyramid, the apex or peak enjoys a nice view supported by the lower blocks, the bottom most layer bears all the weight

the trap of mlm and why it exists is that the perspicacious blocks at the top tap into a very compedent workforce.....housewives............. blow a bit of smoke and mirrors to convince them that some are making money and indeed they are at the top........................

the trap is that a huge amount of quality work goes for tiny returns.


unvaccinated populations subsume vaccinated populations



plus it puts hydrogen in the house water which is an antioxidant

i have a 4300 liter (4.3 cubic meters, 946 UK gallons) corroding galvanized house water tank and using a largish .8mm thick, flat rectangular 316? stainless steel anode, pump about 300ma  (22ma per square meter of cathode/tank wall)  at 18 volts DC in continually from a switched mode plug pak

update 2nd july 2014;  increased the impress voltage to 32v, 410 to 470ma ,  (33ma per square meter of cathode/tank wall) ,  -933 mv with respect to the ref anode ,  -2.2 wrt AG/AgCl ?

the advantages of the increased voltage is the tank water becomes very clear after several days and also puts more hydrogen gas into the water which may be a health benefit !

if the tank is filled and run several days on 32 volts before going away for a while, the impress voltage could be lowered to 12 or 15 which would be adequate to completely prevent corrosion, 12 volts in fact ! ?

you cannot have the anode connected to a copper wire under the water as the copper seems to act as a strong anode to the stainless steel and weakens very quickly !

the 316? stainless steel anode itself is showing no signs of corrosion !

since reference voltages are somewhat indeterministic in this situation i sanded to make a small clear coupon on the lead solder sealing line around the middle of the inside of the tank  (the soldering is done from the tank outside so i was lightly sanding the inside tank wall and not the solder!)  as the most extreme case of corrosion potential and adjusted the voltage/current so as there is absolutely no corrosion and in fact take the cathode potential down even further as with the tank being mild steel and not having any plastic deformation, there is no danger of hydrogen embrittlement !

since at this potential you are getting a reasonable amount of hydrogen being formed on the tank walls, there may be some benefit from getting hydrogen gas into the house water as it is well absorbed through the skin and has an anti-oxidant function in the body !

this low Eh also keeps any lead from forming soluble corrosion products !

for those with the misfortune to have lead soldered stainless steel tanks, this impressed current system would i am sure would stop lead getting into the water !



  YEAH !

first time i had really looked at the issue as one of my car battery terminals is not sealing properly around the base of the post and sulphuric acid is getting onto the flange and making white powder

i tried to dust it off first and thats as a big mistake as it puts powder into the air, i should have wet wiped it

think i must have breathed a bit as am a bit irritable and low serotinin today, though i did eat some cheese for the calcium later to help inhibit the absorbtion of any lead i might have breathed in

so i cleaned the terminals with a sodium barcarbonate solution then mixed 1 part sodium bicarbonate in with 2 parts of lithium grease and smeared that over the the area where the steel screw was touching the lead fitting, as its the dissimilar metals touching, as well as the sulphuric acid that is making the powder

this has worked really well (a year and longer later)



this is what has worked for me on a 3.5 hp side valve briggs and stratton  (classic ?)  mower engine,   try at your own risk !

there's two principles, that of unshrouding the ground electrode so you get better outwards flame kernel propagation and that of reducing the top surface area of the center electrode.

a good  page  explaining some of the theory

the center electrode i filed to roughly an  equilateral triangle  as seen from the top

to orient this triangle, make one side (which we will call the  base) at right angles to the ground electrode line and also nearest to where the ground electrode joins the plug body!

an alternative to filing the center electrode to a triangle is to simply file the back 1/3 of the center electrode off, that is, where it faces the start of the ground electrode on the plug body and just leave the rest circular

this has worked well on a mower that the spark plug was indexed on, presumably this has the effect of moving the spark a bit forward, more away from any shrouding by the ground electrode !

file the end of the ground electrode to a V so that the tip is slightly interior to the tip of the V of the center electrode

i also  flatten  the ground electrode tip slightly

this  “blunting”  of the ground electrode tip seems to have worked quite well

i also file a bit off the top of the ground electrode sloping towards the tip of the V to reduce the mass !

the other ends of the V, that is, where the V stops and the parallel sides of the flat ground electrode begin again should be slightly exterior of the base of the center electrode triangle as seen from above

basically the edges of the V of the ground electrode should very roughly match those of the two non base sides of the center electrode

to file effectively i had to straighten out the ground electrode to make for easier access filing both electrodes, afterwards i reset the gap to 49 thou and this seems to be ok

this gap and indexing seems to deliver very good fuel economy (15 -20% improvement ?) when the engine and presumably plug are hot

the motor starts first or second pull cold

i have the armature air gap so fine that it's actually slightly touching cold and the engine has to get a bit warm to have a full 360º air gap.

i have the armature air gap so close because more energy is transfered, useful for weak flywheel magnets !

not so much as to physically weaken it though, leave plenty of meat !

this system of filing allows for further filing loss while keeping the same dynamic of shapes as the electrode edges may need sharpening up with use!

hopefully the plugs should last quite bit longer than with  side  gapping!










flats !

after filing i dip and swish the end of the plug in a small bottle of meths i keep for that purpose to wash the the filings off !

why this engine has been so responsive despite a very centrally located spark plug is that not having overhead valves or a combustion chamber that forces good mixing of the air/fuel mixture you have a very vertically laminar air/fuel mix column that a combination of indexing and more efficient electrodes gives a good response to, greatly improving the firing of very lean air/fuel ratios on starting for instance !

filed  plug

for car engines i would go to   iridium  as it's too much work to to this on a lot of plugs, indexing especially sucks as filing copper washers takes quite a bit of time!

in reply to what the guy says at the end of the video, the improvement is not so odd, the petrol vapour is never evenly distibuted in the cylinder and the iridium plug gives a more all round ignition wave propogation

replacing the washer that was already on the plug with the copper washer also makes the plug a bit cooler imo as the heat conduction from the plug to the head is better, the plug does seem to take longer to heat up, but the electrode shape modifications discussed above seem to raise the plug tip temperature after 5 minutes running enough !

also since the indexing washer is thinner than the normal washer the plug goes a bit deeper into the cyclinder and more thread area is exposed acting as more of a heatsink !

in this case the extra depth is about .6ml which is not that significant !

i have indexed this plug with the open gap  30º  from the centerpoint between the inlet and exhaust valves towards the inlet valve, but  45º  on a friends mower with a  45  thou gap worked well !

to get this angle i used a store bought a .9 mm thick copper washer and to get the needed extra .08 of a mm made a shim washer out of a beer can wall, drilling it first at slow speed with a 9 mm  (not 10 mm !)  drill bit then using a fine grinding cone on the drill to grind the hole out to 14 mm  (alternating sides !)  and then cutting the washer out with tin snips !

this beer can shim washer is placed between the copper washer and engine so it compresses into a good seal

looking at the clean area versus sooty on the insulator, the midpoint of the clean area aligns with the plug gap direction !

i did see a page that had a  40º  angle towards the inlet valve from the centerpoint between the valves

a good stress test for starting is to not have much fuel in the fuel tank so the air to fuel ratio is greater !

i only did this because my mower had become hard to start and there were no ngk iridium double fine electrode plugs available in that size !






matter !

the way the spark plug lead on many mowers is routed, that is, directly touching metal is problematic and the heat of fins eventually damages any cable directly touching it, also providing an earth to the leakage path !

spark plug leads should be clear of any metal, i cut an oversize hole in the cover and suspended the cable through the center with a bit of plastic cut from the wall of a clear plastic bottle and glued in with a contact adhesive !

PET (recycle number 1) seems to be ok for this  (comparative  dielectric  strengths, table 2/3 the way down the page)

i always keep my head well clear of the flywheel and armature when mower is going to minimise  magnetic  fields damaging the DNA in the brain !


this mower has a plastic bodied carb  (no. 795477)  and the warping of the plastic body is an issue, making diaphragm sealing difficult !

the symptom of this is an extremely rich mixture and the plug gets very sooty within minutes and has to be cleaned using  P240  W&D  sandpaper

also the jet filter wants to be hard up against the intake to restrict the flow into the jet as much as possible as it tends to run rich !

a possibility with the jet is to heat the jet end and squeeze to narrow the orifice?

the problem area is the locus between several chambers and one small valve hole where the distance between them is about 3mm across the flat aluminium so sealing across this is apparently problematic !

i sanded the small valve hole in this area on the plastic with P240 wet and dry  (but using dry !)  and also the other small valve hole in the aluminium and i think that's helped !

why sanding the small valve holes might have worked is dimpling increases the length of surface the petrol has to move over to escape compared to mirror flat!

i also scribed with parallel lines the locus separation areas on top of sanding, maybe the scribing is not necessary? and will give a light sand with P240 when i next have to pull it apart? or just leave the scribe mark as seems to be working?

i had filed the plastic body previously as flat as i could tell  ( a mistake?)

i use the loctite no. 3 non-hardening sealant in a "medium thin" coat on the flat aluminium tank surface in the locus problem area and a dab across the other side to "stick" the diaphragm so it stays in position during futher assembly !

the  #3  should be well shaken before using as it can possibly separate a bit if left standing for months and also allowed to air dry for 5 minutes when applied so it becomes tacky !

cotton buds seem clean out any  #3  fallen into wells or chambers !

permatex have a similar product to loctite!

i did not coat the diaphragm at all !

i then gently tightened it all up taking care not to over torque

this seems to have worked , but i store the mower with the tank empty, either run out or siphoned and press the red primer knob a couple of times so that theres no petrol left in the carb to possibly revert the loctite no. 3 !

silicon is no good, it just becomes like jelly with the petrol !

2nd jan 2012,  the diaphragm sealant only lasts so long,  (boiling it seems to give some recovery !)  so what seems better is to use an older paper gasket to double up the paper gaskets and replace the nitrile diaphragm quite frequently.

12th june 2013,  only use one cardboard gasket and instead of another use an old diaphragm  (with the actual diaphram part cut out not to interfere with the real diaphragm action)  between the one cardboard gasket and the plastic carb body !

the width of the cardboard gasket varies from .45 to .55mm and the diaphragm is .2mm so the addition of the extra diaphragm is significant !



scott   adams  who does the dilbert cartoons is, shall we say,  on autistic spectrum  and regressing in a major way as he gets older !

anyway he became partly non verbal and its interesting to read how he worked around it and i suspect many nonverbal kids are better approached to speaking by rhyme and singing


Good News Day

As regular readers of my blog know, I lost my voice about 18 months ago. Permanently. It's something exotic called Spasmodic Dysphonia. Essentially a part of the brain that controls speech just shuts down in some people, usually after you strain your voice during a bout with allergies (in my case) or some other sort of normal laryngitis. It happens to people in my age bracket.

I asked my doctor – a specialist for this condition – how many people have ever gotten better. Answer: zero. While there's no cure, painful Botox injections through the front of the neck and into the vocal cords can stop the spasms for a few months. That weakens the muscles that otherwise spasm, but your voice is breathy and weak.

The weirdest part of this phenomenon is that speech is processed in different parts of the brain depending on the context. So people with this problem can often sing but they can't talk. In my case I could do my normal professional speaking to large crowds but I could barely whisper and grunt off stage. And most people with this condition report they have the most trouble talking on the telephone or when there is background noise. I can speak normally alone, but not around others. That makes it sound like a social anxiety problem, but it's really just a different context, because I could easily sing to those same people.

I stopped getting the Botox shots because although they allowed me to talk for a few weeks, my voice was too weak for public speaking. So at least until the fall speaking season ended, I chose to maximize my onstage voice at the expense of being able to speak in person.

My family and friends have been great. They read my lips as best they can. They lean in to hear the whispers. They guess. They put up with my six tries to say one word. And my personality is completely altered. My normal wittiness becomes slow and deliberate. And often, when it takes effort to speak a word intelligibly, the wrong word comes out because too much of my focus is on the effort of talking instead of the thinking of what to say. So a lot of the things that came out of my mouth frankly made no sense.

To state the obvious, much of life's pleasure is diminished when you can't speak. It has been tough.

But have I mentioned I'm an optimist?

Just because no one has ever gotten better from Spasmodic Dysphonia before doesn't mean I can't be the first. So every day for months and months I tried new tricks to regain my voice. I visualized speaking correctly and repeatedly told myself I could (affirmations). I used self hypnosis. I used voice therapy exercises. I spoke in higher pitches, or changing pitches. I observed when my voice worked best and when it was worst and looked for patterns. I tried speaking in foreign accents. I tried "singing" some words that were especially hard.

My theory was that the part of my brain responsible for normal speech was still intact, but for some reason had become disconnected from the neural pathways to my vocal cords. (That's consistent with any expert's best guess of what's happening with Spasmodic Dysphonia. It's somewhat mysterious.) And so I reasoned that there was some way to remap that connection. All I needed to do was find the type of speaking or context most similar – but still different enough – from normal speech that still worked. Once I could speak in that slightly different context, I would continue to close the gap between the different-context speech and normal speech until my neural pathways remapped. Well, that was my theory. But I'm no brain surgeon.

The day before yesterday, while helping on a homework assignment, I noticed I could speak perfectly in rhyme. Rhyme was a context I hadn't considered. A poem isn't singing and it isn't regular talking. But for some reason the context is just different enough from normal speech that my brain handled it fine.

Jack be nimble, Jack be quick.
Jack jumped over the candlestick.

I repeated it dozens of times, partly because I could. It was effortless, even though it was similar to regular speech. I enjoyed repeating it, hearing the sound of my own voice working almost flawlessly. I longed for that sound, and the memory of normal speech. Perhaps the rhyme took me back to my own childhood too. Or maybe it's just plain catchy. I enjoyed repeating it more than I should have. Then something happened.

My brain remapped.

My speech returned.

Not 100%, but close, like a car starting up on a cold winter night. And so I talked that night. A lot. And all the next day. A few times I felt my voice slipping away, so I repeated the nursery rhyme and tuned it back in. By the following night my voice was almost completely normal.

When I say my brain remapped, that's the best description I have. During the worst of my voice problems, I would know in advance that I couldn't get a word out. It was if I could feel the lack of connection between my brain and my vocal cords. But suddenly, yesterday, I felt the connection again. It wasn't just being able to speak, it was KNOWING how. The knowing returned.

I still don't know if this is permanent. But I do know that for one day I got to speak normally. And this is one of the happiest days of my life. ”



there's a bit of contention as to wether dinosaurs are birds, or birds and dinosaurs shared a common ancestor, i think the evidence is pointing towards sharing a common ancestor

to sequence 68 million year old dna is just so  astounding

dinosaurs, birds and lizards don't have any brown fat so they have trouble keeping warm, its why birds migrate and probably a big factor in the extinction of dinosaurs

white fat is for storage and brown fat is what is burnt for energy, i would assume overweight people are deficient in nutrients that are needed to convert white to brown fat or may even have an altered genetic expression favouring white over brown fat

brown fat helps newborn and adult mammals maintain their body temperature by burning fat, which converts into heat. The protein UCP1 (Uncoupling Protein 1) has a key role in this energy conversion, which takes place in the cell mitochondria

interestingly pigs have lost this protein  study

"Piglets appear to lack brown adipose tissue, a specific type of fat that is essential for nonshivering thermogenesis in mammals, and they rely on shivering as the main mechanism for thermoregulation. Here we provide a genetic explanation for the poor thermoregulation in pigs as we demonstrate that the gene for uncoupling protein 1 (UCP1) was disrupted in the pig lineage. UCP1 is exclusively expressed in brown adipose tissue and plays a crucial role for thermogenesis by uncoupling oxidative phosphorylation."



i must have low levels of the angiotensin converting enzyme

can't put on fat if i tried, have lowish blood pressure and sure burn through sugar


Australian scientists believe they may have discovered how to help people lose weight without cutting back on food.

Researchers in Melbourne found that by manipulating fat cells in mice they were able to speed up metabolism.

After removing a particular enzyme, scientists found the mice were able to eat the same amount as others but burn more calories and gain less weight.

The breakthrough could pave the way for fat-burning drugs and also help to combat diabetes.

The research found that mice in which the angiotensin converting enzyme (ACE) had been removed were, on average, 20% lighter than normal mice and had up to 60% less body fat.

Because of their faster metabolisms, it also appeared they had less chance of developing diabetes because they processed sugar more quickly.

Drugs which impair the action of ACE in humans already exist, and are used to combat high blood pressure.



i'm in the clear, i'm not a woman and i don't have a waist ;o)

its real interesting actually

what they are saying is that since the number of abdominal fat cells remains constant after adolescence

then more girth on the waist is due to those fat cells getting bigger

this increase in the size of fat cells is the real signaller for increased type 2 diabetes risk (insulin resistance)

its odd, i eat huge amounts of fat yet have practially no fat on me at all and i have always been pretty much this way

the latest research is showing that it's the increase in fat cell size thats very problematic so increasing abdominal fat given that the number of cells stay relatively constant is a very good marker of increasing cell size which is why "pear shaped" is such telling symptom of metabolic syndrome

com'n fatties, fess up


Size of fat cells and waist size predict type 2 diabetes in women  (october 2009)

When it comes to assessing risk for type 2 diabetes, not only do waistlines matter to women, but so does the size of their fat cells. This new discovery helps explain why some women of normal weight develop type 2 diabetes, despite not having any known risk factors.

“Increased knowledge of the link between enlarged fat cells and the development of type 2 diabetes may give rise to new preventive and therapeutic alternatives. Our research also identifies the ratio waist-to-height, waist circumference divided by body height, as a simple tool that can be used to identify women at risk of developing type 2 diabetes.”

The data for this discovery were obtained as part of the "Prospective Study of Women in Gothenburg," performed in Sweden and started in 1968 . For this study, a team of Swedish researchers invited women to free health examinations over the course of 25 years. In 1974-1975, scientists collected abdominal fat biopsies from some of the women and tracked who developed type 2 diabetes. They found that the number of abdominal fat cells remained relatively constant in women after adolescence, but the size of fat cells could change considerably throughout life and were larger in women with type 2 diabetes. In addition, they found that waist-to-height ratio may also be a good indicator of diabetes risk.

According to the U.S. Centers for Disease Control and Prevention, type 2 diabetes may account for 90 to 95 percent of all diagnosed cases of diabetes. The disease begins as insulin resistance, and as the need for insulin rises, the pancreas gradually loses its ability to produce insulin. Type 2 diabetes often is associated with older age, but is increasingly being diagnosed in children. Obesity, family history of diabetes, history of gestational diabetes, impaired glucose metabolism, physical inactivity, and race/ethnicity also play a role in whether or not someone develops the disease. In particular, African Americans, Hispanic/Latino Americans, American Indians/Native Americans, and some Asian Americans and Native Hawaiians or Other Pacific Islanders are at high risk for type 2 diabetes.



obesity is a condition of trace mineral malnutrition, biofilm toxins and a lack of skin vitamin D which is addressed by the compendium™ and BCD™

its really a symptom and can't be addressed directly, as you can see from the study below estimating 25% of the bodies genes have an impact on weight

thats basically why all the weight loss programs are fools gold, no shortage of those ;o)

the compendium and BCD do give an excellent and optimal body shape, the roots of the biofilm carbohydrate diet go back to the early greek games and the very first olympics in 776 BC as an athletes diet

weight problems should be judged by looking at the body shape and bone structure, overweight should always be judged visually like this and never be checked by weight

skinny is the bodies response to reducing the metabolic load, so you have to look at the reason why and work from causes and not attempt to build up weight or muscle mass directly as this just increases the metabolic load which the body couldn't cope with in the first place, muscle is especially very metabolically expensive as you may notice with all the brain dead and joint damaged bodybuilders


PHILADELPHIA (January 14, 2008) – Reporting in the online journal BMC Genetics, researchers from the Monell Center have for the first time attempted to count the number of genes that contribute to obesity and body weight.

The findings suggest that over 6,000 genes – about 25 percent of the genome – help determine an individual's body weight.

"Reports describing the discovery of a new `obesity gene' have become common in the scientific literature and also the popular press," notes Monell behavioral geneticist Michael G. Tordoff, PhD, an author on the study.

"Our results suggest that each newly discovered gene is just one of the many thousands that influence body weight, so a quick fix to the obesity problem is unlikely."

To obtain an estimate of how many genes contribute to body weight, the Monell researchers surveyed the Jackson Laboratory Mouse Genome Database for information on body weights of knockout mouse strains.

Knockout mice have had a specific gene inactivated, or "knocked out." By studying how the knockout mice differ from normal mice, researchers obtain information about that gene's function and how it might contribute to disease. Mice can provide valuable information on human disease because they share many genes with humans.

The knockout approach is so useful that the inventors of the technology were awarded the 2007 Nobel Prize in Medicine. Knockout mice are now standard tools in all mouse models of behavior and disease.

In 60% of strains, knocking out a gene produces mice that are nonviable; that is, the mouse cannot survive without the knocked out gene.

The Monell survey revealed that body weight was altered in over a third of the viable knockout stains; 31 percent weighed less than controls (indicating that the missing genes contribute to heavier body weight), while another 3 percent weighed more (contributing to lighter weight).

Extrapolating from the total number of genes in the mouse genome, this implies that over 6,000 genes could potentially contribute to the body weight of a mouse.

Tordoff comments, "It is interesting that there are 10 times more genes that increase body weight than decrease it, which might help explain why it is easier to gain weight than lose it."

Because body weight plays a role in many diseases, including hypertension, diabetes, and heart disease, the implications of the findings extend beyond studies of obesity and body weight. Gene knockouts reported to affect these diseases and others could potentially be due to a general effect to lower body weight.

The findings also hold clinical relevance, according to lead author Danielle R. Reed, PhD, a Monell geneticist.

“ Clinicians and other professionals concerned with the development of personalized medicine need to expand their ideas of genetics to recognize that many genes act together to determine disease susceptibility. ”



obesity is a condition of malnutrition as well as being partly genetic

the genetic side makes it not very tractable if you (the obese!) remain in your present dietary and nutritional space, especially vitamin D deficiency

if you want to get out you have to traverse to that super metabolic space your body and biochemistry will inhabit on the full compendium which includes adequate skin vitamin D

the full compendium and BCD can give a superbly shaped body, interestingly, the SCD and BCD have their origins going back to the original greek olympics as an athletes diet


what a man looked like 2400 years ago


bit fat faced actually - he's in very good condition, but he may well have been doing some equivalent of the BCD since atheletes did do that sort of diet in those times, not the carb loading and steriods favoured now

it just shows you how much of the athletic musculature these days is steriodal


obesity can cause type 2 diabetes

c'mon fatties!

a huge incentive not to be obese, the below is from a monash university study (july 2009) led by matthew watt

“ fat cells release a novel protein called PEDF (pigment epithelium-derived factor), which triggers a chain of events and interactions that lead to development of type 2 diabetes.

when PEDF is released into the bloodstream, it causes the muscle and liver to become desensitised to insulin. the pancreas then produces more insulin to counteract these negative effects

this insulin release causes the pancreas to become overworked, eventually slowing or stopping insulin release from the pancreas, leading to Type 2 diabetes.

it appears that the more fat tissue a person has the less sensitive they become to insulin. therefore a greater amount of insulin is required to maintain the body's regulation of blood-glucose

our research was able to show that increasing PEDF not only causes type 2 diabetes like complications but that blocking PEDF reverses these effects. the body again returned to being insulin-sensitive and therefore did not need excess insulin to remain regulated.

identifying the link is a significant breakthrough in explaining the reasons why obesity triggers the onset of Type 2 Diabetes. ”

let me guess the  ohhh i can't change anything  and not doing anything effective to ensure that

i am all for weight proportional airfares the attitude of the fat is so pissy!

obesity is a condition of malnutrition, the same mineral shortages that give rise to a depressed metabolism also stress the pancreas and limit it's ability to produce insulin as well as increasing insulin resistance

the lack of sun on the skin also has a role !

obesity is nothing to do with exercise ! exercise will not fix a depressed metabolism, just create a different set of problems !

  “ osteocalcin, a protein produced by bone cells, has an impact not only on bone but also on fat tissue metabolism. The protein's function is associated with bone formation and bone mineralization. But recent data suggest that osteocalcin is also involved in the regulation of glucose and fat metabolism and that osteocalcin levels are  lower  in obese and overweight individuals ”

vitamin K in combination with vitamin D promote the production of osteocalcin by the osteoblasts

interestingly, warfarin which  blocks  vitamin K, causes weight gain and loss of insulin sensitivity !

if osteocalcin is low you have weight gain with a loss of bone strength and some aspects of metabolic syndrome like lower insulin sensitivity which is a cumulatively destructive combination !

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